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Traumatic brain injury (TBI) is a serious problem causing mortality and morbidity in children and adolescents. Hemorrhage, edema, and penetrating injury in the brain parenchyma are considered as important mechanisms of TBI. Neuron-specific enolase and leukocyte common antigen are validated biomarkers for TBI. Both biomarkers could not differentiate TBI at different locations in the brain, although the elevation of both markers may be significant. The primary objective of the present study was to evaluate the relationship between TBI and the location of TBI in relation to the brain parenchyma using neuronal specific enolase and leukocyte common antigen. A total of 50 children with a TBI diagnosis on the basis of findings on computed tomography and/or cerebral tomography and neurosurgeon were enrolled. Their age range was from the first day of admission to the last day of admission. Leukocyte common antigen and neuron-specific enolase were analyzed on day 1, 3, 5, and 7 after TBI, and any relationship between the levels of these proteins and the location of TBI in relation to the brain parenchyma was studied. Neuronal specific enolase values were the highest in patients with injury in the anterior and middle skull base. Neuron-specific enolase levels in children with penetrating brain injury were observed to be higher than those with extra-axial hemorrhage and those with no evidence of injury on head computed tomography. Leukocyte common antigen levels were relatively lower than those of the neuron-specific enolase. In conclusion, neuron-specific enolase levels were higher in patients with TBI in the anterior and middle skull base. However, leukocyte common
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